Cancer Drug Shortfalls Tied to How BET Inhibitors Hit BRD2 and BRD4 Differently
Using rapid protein degradation, chemogenomics, and super‑resolution microscopy in mouse embryonic stem cells, the team dissected the distinct contributions of BRD2 and BRD4 to transcription. The post Cancer Drug Shortfalls Tied to How BET Inhibitors Hit BRD2 and BRD4 Differently appeared first on GEN - Genetic Engineering and Biotechnology News.
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